Small Molecule Mitochondrial F1F0 ATPase Hydrolase Inhibitors as Cardioprotective Agents. Identification of 4-(N-Arylimidazole)-Substituted Benzopyran Derivatives as Selective Hydrolase Inhibitors-Reference-Cited by-同舟云学术

Small Molecule Mitochondrial F1F0 ATPase Hydrolase Inhibitors as Cardioprotective Agents. Identification of 4-(N-Arylimidazole)-Substituted Benzopyran Derivatives as Selective Hydrolase Inhibitors

Published:2004-01-29 Issue:5 Volume:47 Page:1081-1084
ISSN:0022-2623
Container-title:Journal of Medicinal Chemistry
language:en
Short-container-title:J. Med. Chem.

Author:

Atwal Karnail S.¹,Wang Paulina¹,Rogers W. Lynn¹,Sleph Paul¹,Monshizadegan Hossain¹,Ferrara Francis N.¹,Traeger Sarah¹,Green David W.¹,Grover Gary J.¹

Affiliation:

1. The Bristol-Myers Squibb Pharmaceutical Research Institute, P.O. Box 5400, Princeton, New Jersey 08543-5400

Publisher

American Chemical Society (ACS)

Subject

Drug Discovery,Molecular Medicine

Link

https://pubs.acs.org/doi/pdf/10.1021/jm030291x

Reference18 articles.

1. Effect of inhibition of the mitochondrial ATPase on net myocardial ATP in total ischemia

2. Structure at 2.8 Â resolution of F1-ATPase from bovine heart mitochondria

3. Control of mitochondrial ATP synthesis in the heart

Cited by 42 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Hydrolytic Activity of Mitochondrial F₁F_O-ATP Synthase as a Target for Myocardial Ischemia–Reperfusion Injury: Discovery and In Vitro and In Vivo Evaluation of Novel Inhibitors;Journal of Medicinal Chemistry;2023-11-09

2. F1Fo adenosine triphosphate (ATP) synthase is a potential drug target in non-communicable diseases;Molecular Biology Reports;2023-01-30

3. The F1Fo-ATPase inhibitor protein IF1 in pathophysiology;Frontiers in Physiology;2022-08-04

4. Sirtuin 5 depletion impairs mitochondrial function in human proximal tubular epithelial cells;Scientific Reports;2021-07-30

5. New Mechanistic Insights on Carbon Nanotubes’ Nanotoxicity Using Isolated Submitochondrial Particles, Molecular Docking, and Nano-QSTR Approaches;Biology;2021-02-25