Hypoxia and Reoxygenation Induce Endothelial Nitric Oxide Synthase Uncoupling in Endothelial Cells through Tetrahydrobiopterin Depletion and S-Glutathionylation-Reference-Cited by-同舟云学术

Hypoxia and Reoxygenation Induce Endothelial Nitric Oxide Synthase Uncoupling in Endothelial Cells through Tetrahydrobiopterin Depletion and S-Glutathionylation

Published:2014-05-29 Issue:22 Volume:53 Page:3679-3688
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

De Pascali Francesco¹,Hemann Craig¹,Samons Kindra¹,Chen Chun-An¹²,Zweier Jay L.¹

Affiliation:

1. Davis Heart and Lung Research Institute and Division of Cardiovascular Medicine, Department of Internal Medicine, College of Medicine, The Ohio State University, Columbus, Ohio 43210, United States

2. The Department of Emergency Medicine, College of Medicine, The Ohio State University, Columbus, Ohio 43210, United States

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi500076r

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2. Prognostic Impact of Coronary Vasodilator Dysfunction on Adverse Long-Term Outcome of Coronary Heart Disease

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4. New insights on myocardial pyridine nucleotides and thiol redox state in ischemia and reperfusion damage

5. Redox proteomics of thiol proteins in mouse heart during ischemia/reperfusion using ICAT reagents and mass spectrometry

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