The Hypervariable Region of K-Ras4B Is Responsible for Its Specific Interactions with Calmodulin-Reference-Cited by-同舟云学术

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The Hypervariable Region of K-Ras4B Is Responsible for Its Specific Interactions with Calmodulin

Published:2009-07-23 Issue:32 Volume:48 Page:7575-7583
ISSN:0006-2960
Container-title:Biochemistry
language:en
Short-container-title:Biochemistry

Author:

Abraham Sherwin J.¹,Nolet Ryan P.¹,Calvert Richard J.²³,Anderson Lucy M.²,Gaponenko Vadim¹

Affiliation:

1. Department of Biochemistry and Molecular Genetics, University of Illinois, Chicago, Illinois 60607

2. Laboratory of Comparative Carcinogenesis, National Cancer Institute, Frederick, Maryland 21702

3. Division of Bioanalytical Chemistry, U.S. Food and Drug Administration, College Park, Maryland 20740

Publisher

American Chemical Society (ACS)

Subject

Biochemistry

Link

https://pubs.acs.org/doi/pdf/10.1021/bi900769j

Reference34 articles.

2. Activation of ras Oncogenes Preceding the Onset of Neoplasia

3. Mouse model for lung tumorigenesis through Cre/lox controlled sporadic activation of the K-Ras oncogene

4. Targeted expression of oncogenic K-ras in intestinal epithelium causes spontaneous tumorigenesis in mice

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1. K-Ras Binds Calmodulin-Related Centrin1 with Potential Implications for K-Ras Driven Cancer Cell Stemness;Cancers;2023-06-07

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3. Novel approaches for the development of direct KRAS inhibitors: structural insights and drug design;Expert Opinion on Drug Discovery;2022-01-27

4. The dynamic nature of the K-Ras/calmodulin complex can be altered by oncogenic mutations;Current Opinion in Structural Biology;2021-12

5. Calmodulin extracts the Ras family protein RalA from lipid bilayers by engagement with two membrane-targeting motifs;Proceedings of the National Academy of Sciences;2021-09-03

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