ClC-3 Is an Intracellular Chloride/Proton Exchanger with Large Voltage-Dependent Nonlinear Capacitance
Author:
Affiliation:
1. Institut für Neurophysiologie, Medizinische Hochschule Hannover, D-30625 Hannover, Germany
2. Institute of Complex Systems, Zelluläre Biophysik, Forschungszentrum Jülich, 52425 Jülich, Germany
Publisher
American Chemical Society (ACS)
Subject
Cell Biology,Cognitive Neuroscience,Physiology,Biochemistry,General Medicine
Link
https://pubs.acs.org/doi/pdf/10.1021/cn400032z
Reference58 articles.
1. CLC Chloride Channels and Transporters: From Genes to Protein Structure, Pathology and Physiology
2. Cloning and expression of a protein kinase C-regulated chloride channel abundantly expressed in rat brain neuronal cells
3. Disruption of ClC-3, a Chloride Channel Expressed on Synaptic Vesicles, Leads to a Loss of the Hippocampus
4. Presynaptic CLC-3 determines quantal size of inhibitory transmission in the hippocampus
5. Altered GABAergic function accompanies hippocampal degeneration in mice lacking ClC-3 voltage-gated chloride channels
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