Highly Selective Artificial K<sup>+</sup> Transporters Reverse Liver Fibrosis In Vivo-Reference-Cited by-同舟云学术

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Highly Selective Artificial K⁺ Transporters Reverse Liver Fibrosis In Vivo

Published:2024-08-26 Issue: Volume: Page:
ISSN:2691-3704
Container-title:JACS Au
language:en
Short-container-title:JACS Au

Author:

Zhang Qiuping¹²,Liang Qinghong¹²,Wang Guijiang¹,Xie Xiaopan¹,Cao Yin¹,Sheng Nan¹,Zeng Zhiping¹,Ren Changliang¹²^ORCID

Affiliation:

1. State Key Laboratory of Cellular Stress Biology and Fujian Provincial Key Laboratory of Innovative Drug Target Research, School of Pharmaceutical Sciences, Xiamen University, Xiamen, Fujian 361102, China

2. Shenzhen Research Institute of Xiamen University, Shenzhen, Guangdong 518057, China

Funder

Science, Technology and Innovation Commission of Shenzhen Municipality

National Natural Science Foundation of China

Nanqiang Youth Scholar Program of Xiamen University

Publisher

American Chemical Society (ACS)

Link

https://pubs.acs.org/doi/pdf/10.1021/jacsau.4c00521

Reference56 articles.

1. Ionic Channels in Biological Membranes: Natural Nanotubes

2. Na+,K+-ATPase as a Target for Treatment of Tissue Fibrosis

3. Activation of BK Channels Prevents Hepatic Stellate Cell Activation and Liver Fibrosis Through the Suppression of TGFβ1/SMAD3 and JAK/STAT3 Profibrotic Signaling Pathways

4. The roles of K+ channels in cancer

5. Up-regulated expression of two-pore domain K+ channels, KCNK1 and KCNK2, is involved in the proliferation and migration of pulmonary arterial smooth muscle cells in pulmonary arterial hypertension

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