Inhibition of NRF2 transcription factor mediated by MIR-155 diminishes melanoma cell viability independently of redox status

Author:

Kutsenko V. A.1,Dashkova D. A.1,Ruksha T. G.1

Affiliation:

1. Voino-Yasenetsky Krasnoyarsk State Medical University

Abstract

Redox-sensitive NRF2 transcription factor is a target gene of microRNA miR-155. miR-155 mimic was transfected in dacarbazine-resistant melanoma cells. NRF2 expression levels were down-regulated in miR-155-overexpressed cells independently of oxidative stress induced by hydrogen peroxide. NRF2 suppression was associated with a decrease of melanoma cells viability. As a result, miR-155-mediated NRF2 overexpression that regulate intensity of a cell antioxidant processes can be associated with cancer cell survival leading to drug resistance. NRF2 repression by miR-155 highlighted a potential for NRF2 down-regulation as an approach in anticancer therapy.

Publisher

The Russian Academy of Sciences

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