S100A9 and SOCS3 as diagnostic biomarkers of acute myocardial infarction and their association with immune infiltration
Author:
Affiliation:
1. Department of Emergency Medicine, Tianjin Medical University General Hospital,
Publisher
Genetics Society of Japan
Subject
Genetics,Molecular Biology,General Medicine
Link
https://www.jstage.jst.go.jp/article/ggs/97/2/97_21-00073/_pdf
Reference81 articles.
1. Averill, M. M., Kerkhoff, C., and Bornfeldt, K. E. (2012) S100A8 and S100A9 in cardiovascular biology and disease. Arterioscler. Thromb. Vasc. Biol. 32, 223–229.
2. Backteman, K., Ernerudh, J., and Jonasson, L. (2014) Natural killer (NK) cell deficit in coronary artery disease: no aberrations in phenotype but sustained reduction of NK cells is associated with low-grade inflammation. Clin. Exp. Immunol. 175, 104–112.
3. Braunwald, E. (2012) Unstable angina and non-ST elevation myocardial infarction. Am. J. Respir. Crit. Care Med. 185, 924–932.
4. Browne, M. W. (2000) Cross-validation methods. J. Math. Psychol. 44, 108–132.
5. Cai, Z., Xie, Q., Hu, T., Yao, Q., Zhao, J., Wu, Q., and Tang, Q. (2020) S100A8/A9 in myocardial infarction: a promising biomarker and therapeutic target. Front. Cell Dev. Biol. 8, 603902.
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