The comparison of intrinsic rates of increase among chromosome-substituted lines resistant and susceptible to organophosphate insecticides in Drosophila melanogaster.
Author:
Affiliation:
1. Institute of Cell, Animal and Population Biology, Ashworth Laboratories, University of Edinburgh
2. Institute of Biological Sciences, University of Tsukuba
Publisher
Genetics Society of Japan
Subject
Genetics,Molecular Biology,General Medicine
Link
http://www.jstage.jst.go.jp/article/ggs/78/5/78_5_373/_pdf
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3. Caswell, H. (2001) Matrix population models, 2nd edn. Sinauer Associates, Sunderland, Massachusetts, USA.
4. Selection in populations with overlapping generations. III. Conditions for genetic equilibrium
5. Evolutionary mechanisms of senescence
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1. Population model of fluctuations in organophosphate resistance of Drosophila melanogaster: Roles of a mutated acetylcholinesterase and a cytochrome P450;Russian Journal of Ecology;2011-10-25
2. Misconceptions about the comparison of intrinsic rates of natural increase;Journal of Applied Entomology;2011-01-27
3. Contributions of three-site mutations in acetylcholinesterase and cytochrome P450 to genetic variation in susceptibility to organophosphate insecticides within a natural population of Drosophila melanogaster;Population Ecology;2009-05-02
4. The DHR96 nuclear receptor regulates xenobiotic responses in Drosophila;Cell Metabolism;2006-07
5. Seasonal fluctuation in susceptibility to insecticides within natural populations of Drosophila melanogaster. II. Features of genetic variation in susceptibility to organophosphate insecticides within natural populations of D. melanogaster;Genes & Genetic Systems;2006
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