Intermittent Hypoxia Induces Autophagy to Protect Cardiomyocytes From Endoplasmic Reticulum Stress and Apoptosis-Reference-Cited by-同舟云学术

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Intermittent Hypoxia Induces Autophagy to Protect Cardiomyocytes From Endoplasmic Reticulum Stress and Apoptosis

Published:2019-08-07 Issue: Volume:10 Page:
ISSN:1664-042X
Container-title:Frontiers in Physiology
language:
Short-container-title:Front. Physiol.

Author:

Chang Jui-Chih,Hu Wei-Fen,Lee Wen-Sen,Lin Jian-Hong,Ting Pei-Ching,Chang Huai-Ren,Shieh Kun-Ruey,Chen Tsung-I,Yang Kun-Ta

Funder

Ministry of Science and Technology

Buddhist Compassion Relief Tzu Chi Foundation

Publisher

Frontiers Media SA

Subject

Physiology (medical),Physiology

Reference48 articles.

1. Endoplasmic reticulum stress as a novel inducer of hypoxia inducible factor-1 activity: its role in the susceptibility to myocardial ischemia-reperfusion induced by chronic intermittent hypoxia;Belaidi;Int. J. Cardiol.,2016

2. High-intensity training reduces intermittent hypoxia-induced ER stress and myocardial infarct size;Bourdier;Am. J. Physiol. Heart Circ. Physiol.,2016

3. Endoplasmic reticulum stress plays critical role in brain damage after chronic intermittent hypoxia in growing rats;Cai;Exp. Neurol.,2014

4. Non-lethal levels of oxidative stress in response to short-term intermittent hypoxia enhance ca2+ handling in neonatal rat cardiomyocytes;Chen;Cell. Physiol. Biochem.,2014

5. Role of sodium-hydrogen exchanger-1 (NHE-1) in the effect of exercise on intermittent hypoxia-induced left ventricular dysfunction;Chen;Chin. J. Physiol.,2015

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