Herpes Simplex Virus 1-Induced Blood-Brain Barrier Damage Involves Apoptosis Associated With GM130-Mediated Golgi Stress-Reference-Cited by-同舟云学术

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Herpes Simplex Virus 1-Induced Blood-Brain Barrier Damage Involves Apoptosis Associated With GM130-Mediated Golgi Stress

Published:2020-01-24 Issue: Volume:13 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

He Qiang,Liu Hui,Huang Chuxin,Wang Renchun,Luo Minhua,Lu Wei

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference44 articles.

1. Structure and function of the blood-brain barrier;Abbott;Neurobiol. Dis.,2010

2. The specific fates of tight junction proteins in apoptotic epithelial cells;Bojarski;J. Cell Sci.,2004

3. [18F]FHPG positron emission tomography for detection of herpes simplex virus (HSV) in experimental HSV encephalitis;Buursma;J. Virol.,2005

4. GOLGA2/GM130, cis-Golgi matrix protein, is a novel target of anticancer gene therapy;Chang;Mol. Ther.,2012

5. Disruption of in vitro endothelial barrier integrity by Japanese encephalitis virus-Infected astrocytes;Chang;Glia,2015

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1. Viral Infection and Ischemic Stroke: Emerging Trends and Mechanistic Insights;Journal of the American Heart Association;2024-09-11

2. GM130-silencing may aggravate blood-brain barrier damage and affect microglia polarization by down-regulating PD-L1 expression after experimental intracerebral hemorrhage;Molecular Biology Reports;2024-08-19

3. Pseudorabies virus gM and its homologous proteins in herpesviruses induce mitochondria-related apoptosis involved in viral pathogenicity;PLOS Pathogens;2024-04-26

4. Polyphenols from Maackia amurensis Heartwood Protect Neuronal Cells from Oxidative Stress and Prevent Herpetic Infection;International Journal of Molecular Sciences;2024-04-09

5. HSV-1 immune escapes in microglia by down-regulating GM130 to inhibit TLR3-mediated innate immune responses;2024-02-19

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