Single Ethanol Withdrawal Regulates Extrasynaptic δ-GABAA Receptors Via PKCδ Activation-Reference-Cited by-同舟云学术

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Single Ethanol Withdrawal Regulates Extrasynaptic δ-GABAA Receptors Via PKCδ Activation

Published:2018-04-26 Issue: Volume:11 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Chen Juan,He Yang,Wu Yan,Zhou Hang,Su Li-Da,Li Wei-Nan,Olsen Richard W.,Liang Jing,Zhou Yu-Dong,Shen Yi

Funder

National Natural Science Foundation of China

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference57 articles.

1. Alcohol and the brain: neuronal molecular targets, synapses, and circuits;Abrahao;Neuron,2017

2. Protein kinase C phosphorylation regulates membrane insertion of GABAA receptor subtypes that mediate tonic inhibition;Abramian;J. Biol. Chem.,2010

3. Differential regulation of synaptic and extrasynaptic α4 GABAA receptor populations by protein kinase A and protein kinase C in cultured cortical neurons;Bohnsack;Neuropharmacology,2016

4. Decreased ethanol sensitivity and tolerance development in γ-protein kinase C null mutant mice is dependent on genetic background;Bowers;Alcohol. Clin. Exp. Res.,1999

5. Ethanol consumption and behavioral impulsivity are increased in protein kinase Cγ null mutant mice;Bowers;J. Neurosci.,2001

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