ALWPs Improve Cognitive Function and Regulate Aβ Plaque and Tau Hyperphosphorylation in a Mouse Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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ALWPs Improve Cognitive Function and Regulate Aβ Plaque and Tau Hyperphosphorylation in a Mouse Model of Alzheimer’s Disease

Published:2019-08-16 Issue: Volume:12 Page:
ISSN:1662-5099
Container-title:Frontiers in Molecular Neuroscience
language:
Short-container-title:Front. Mol. Neurosci.

Author:

Nam Youngpyo,Joo Bitna,Lee Ju-Young,Han Kyung-Min,Ryu Ka-Young,Koh Young Ho,Kim Jeongyeon,Koo Ja Wook,We Young-Man,Hoe Hyang-Sook

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference73 articles.

1. Phosphorylation of tau at Thr212, Thr231 and Ser262 combined causes neurodegeneration;Alonso;J. Biol. Chem.,2010

2. Putative function of ADAM9, ADAM10, and ADAM17 as APP α-secretase;Asai;Biochem. Biophys. Res. Commun.,2003

3. Depletion of microglia and inhibition of exosome synthesis halt tau propagation;Asai;Nat. Neurosci.,2015

4. Specific tau phosphorylation sites correlate with severity of neuronal cytopathology in Alzheimer’s disease;Augustinack;Acta Neuropathol.,2002

5. Optimized survival of hippocampal neurons in B27-supplemented Neurobasal, a new serum-free medium combination;Brewer;J. Neurosci. Res.,1993

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