CaMKII-dependent responses to ischemia and reperfusion challenges in the heart-Reference-Cited by-同舟云学术

CaMKII-dependent responses to ischemia and reperfusion challenges in the heart

Published:2014-05-06 Issue: Volume:5 Page:
ISSN:1663-9812
Container-title:Frontiers in Pharmacology
language:
Short-container-title:Front. Pharmacol.

Author:

Bell James R.,Vila-Petroff Martin,Delbridge Lea M. D.

Publisher

Frontiers Media SA

Subject

Pharmacology (medical),Pharmacology

Reference54 articles.

1. Sex and sex hormones in cardiac stress-Mechanistic insights.;Bell;J. Steroid Biochem. Mol. Biol.,2013a

2. Augmented CaMKII recruitment despite less reperfusion arrhythmias in female hearts â€“ a matter of differential post-translational modification of CaMKII splice variants?;Bell;Proc. Aust. Physiol. Soc.,2013b

3. Ca2+/calmodulin-dependent protein kinase inhibition suppresses post-ischemic arrhythmogenesis and mediates sinus bradycardic recovery in reperfusion.;Bell;Int. J. Cardiol.,2012

4. Differential post-translational modification of CaMKII splice variants may be critical to determining post-ischemic arrhythmia vulnerability in male and female hearts.;Bell;Global Heart,2014

5. Signal transduction mechanisms involved in cardiac preconditioning: role of Ras-GTPase, Ca2+/calmodulin-dependent protein kinase II and epidermal growth factor receptor.;Benter;Mol. Cell. Biochem.,2005

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1. An improved reporter identifies ruxolitinib as a potent and cardioprotective CaMKII inhibitor;Science Translational Medicine;2023-06-21

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5. The Agonist of Inward Rectifier Potassium Channel (I_K1) Attenuates Rat Reperfusion Arrhythmias Linked to CaMKII Signaling;International Heart Journal;2021-11-29