Antigen Loading (e.g., Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice-Reference-Cited by-同舟云学术

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Antigen Loading (e.g., Glutamic Acid Decarboxylase 65) of Tolerogenic DCs (tolDCs) Reduces Their Capacity to Prevent Diabetes in the Non-Obese Diabetes (NOD)-Severe Combined Immunodeficiency Model of Adoptive Cotransfer of Diabetes As Well As in NOD Mice

Published:2018-02-16 Issue: Volume:9 Page:
ISSN:1664-3224
Container-title:Frontiers in Immunology
language:
Short-container-title:Front. Immunol.

Author:

Funda David P.,Goliáš Jaroslav,Hudcovic Tomáš,Kozáková Hana,Špíšek Radek,Palová-Jelínková Lenka

Funder

Grantová Agentura České Republiky

Publisher

Frontiers Media SA

Subject

Immunology,Immunology and Allergy

Reference55 articles.

1. Insulin-dependent diabetes mellitus as an autoimmune disease;Bach;Endocr Rev,1994

2. Type 1 diabetes – from pathogenesis to prevention;Harrison,1999

3. Mouse models for type 1 diabetes;Van Belle;Drug Discov Today Dis Models,2009

4. The immunotherapeutic potential of dendritic cells in type 1 diabetes;Mukherjee;Clin Exp Immunol,2010

5. Regulatory Th2 response induced following adoptive transfer of dendritic cells in prediabetic NOD mice;Feili-Hariri;Eur J Immunol,2002

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