Molecular Defects in Friedreich’s Ataxia: Convergence of Oxidative Stress and Cytoskeletal Abnormalities-Reference-Cited by-同舟云学术

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Molecular Defects in Friedreich’s Ataxia: Convergence of Oxidative Stress and Cytoskeletal Abnormalities

Published:2020-11-09 Issue: Volume:7 Page:
ISSN:2296-889X
Container-title:Frontiers in Molecular Biosciences
language:
Short-container-title:Front. Mol. Biosci.

Author:

Smith Frances M.,Kosman Daniel J.

Funder

Foundation for the National Institutes of Health

Publisher

Frontiers Media SA

Subject

Biochemistry, Genetics and Molecular Biology (miscellaneous),Molecular Biology,Biochemistry

Reference156 articles.

1. Discovery of direct inhibitors of Keap1–Nrf2 protein–protein interaction as potential therapeutic and preventive agents.;Abed;Acta Pharm. Sin. B,2015

2. Novel Nrf2-inducer prevents mitochondrial defects and oxidative stress in Friedreich’s ataxia models.;Abeti;Front. Cell. Neurosci.,2018

3. Mitochondria contribute to NADPH generation in mouse rod photoreceptors.;Adler;J. Biol. Chem.,2014

4. Exonic deletions of FXN and early-onset Friedreich ataxia.;Anheim;Arch. Neurol.,2012

5. Regulation of mitochondrial iron accumulation by Yfh1p, a putative homolog of frataxin.;Babcock;Science,1997

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1. Unusual Age‐Dependent Behavior of Leukocytes Telomere Length in Friedreich's Ataxia;Movement Disorders;2024-09-05

2. New and Emerging Drug and Gene Therapies for Friedreich Ataxia;CNS Drugs;2024-08-08

3. Exploring mitochondrial biomarkers for Friedreich's ataxia: a multifaceted approach;Journal of Neurology;2024-03-23

4. Loss of filamentous actin, tight junction protein expression, and paracellular barrier integrity in frataxin-deficient human brain microvascular endothelial cells—implications for blood-brain barrier physiology in Friedreich’s ataxia;Frontiers in Molecular Biosciences;2024-01-11

5. Glutathionyl Hemoglobin and Its Emerging Role as a Clinical Biomarker of Chronic Oxidative Stress;Antioxidants;2023-11-07

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