Exogenous Hydrogen Sulfide Offers Neuroprotection on Intracerebral Hemorrhage Injury Through Modulating Endogenous H2S Metabolism in Mice-Reference-Cited by-同舟云学术

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Exogenous Hydrogen Sulfide Offers Neuroprotection on Intracerebral Hemorrhage Injury Through Modulating Endogenous H2S Metabolism in Mice

Published:2019-08-07 Issue: Volume:13 Page:
ISSN:1662-5102
Container-title:Frontiers in Cellular Neuroscience
language:
Short-container-title:Front. Cell. Neurosci.

Author:

Shan Haiyan,Qiu Jianping,Chang Pan,Chu Yang,Gao Cheng,Wang Haocheng,Chen Guang,Luo Chengliang,Wang Tao,Chen Xiping,Zhang Mingyang,Tao Luyang

Funder

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Frontiers Media SA

Subject

Cellular and Molecular Neuroscience

Reference38 articles.

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2. Emboli formation rather than inflammatory mediators are responsible for increased cerebral water content after conventional and assisted beating-heart myocardial revascularization in a porcine model.;Bierbach;Stroke,2008

3. Programmed cell death after intracerebral hemorrhage.;Bobinger;Curr. Neuropharmacol.,2018

4. Autophagy is increased in mice after traumatic brain injury and is detectable in human brain after trauma and critical illness.;Clark;Autophagy,2008

5. IL-33 exerts neuroprotective effect in mice intracerebral hemorrhage model through suppressing inflammation/apoptotic/autophagic pathway.;Gao;Mol. Neurobiol.,2017

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