The Cytoplasmic Region of Plasmodium falciparum SURFIN4.2 Is Required for Transport from Maurer’s Clefts to the Red Blood Cell Surface
Author:
Affiliation:
1. Section of Environmental Parasitology, Graduate School of Medical and Dental Sciences, Tokyo Medical and Dental University
2. Department of Protozoology, Institute of Tropical Medicine (NEKKEN), Nagasaki University
Publisher
Japanese Society of Tropical Medicine
Subject
Infectious Diseases,Public Health, Environmental and Occupational Health
Link
https://www.jstage.jst.go.jp/article/tmh/43/4/43_2015-38/_pdf
Reference38 articles.
1. 1 World Health Organization. World Malaria Report 2014. Geneva: World Health Organization; 2014.
2. 2 Prajapati SK, Culleton R, Singh OP. Protein trafficking in Plasmodium falciparum-infected red cells and impact of the expansion of exported protein families. Parasitology 2014; 141: 1533–1543.
3. 3 de Koning-Ward TF, Gilson PR, Boddey JA, et al. A newly discovered protein export machine in malaria parasites. Nature 2009; 459: 945–949.
4. 4 Beck JR, Muralidharan V, Oksman A, et al. PTEX component HSP101 mediates export of diverse malaria effectors into host erythrocytes. Nature 2014; 511: 592–595.
5. 5 Elsworth B, Matthews K, Nie CQ, et al. Ward TF. PTEX is an essential nexus for protein export in malaria parasites. Nature 2014; 511: 587–591.
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