Luteinizing Hormone Receptor-Stimulated Progesterone Production by Preovulatory Granulosa Cells Requires Protein Kinase A-Dependent Activation/Dephosphorylation of the Actin Dynamizing Protein Cofilin

Author:

Karlsson Amelia B.12,Maizels Evelyn T.1,Flynn Maxfield P.1,Jones Jonathan C.1,Shelden Eric A.2,Bamburg James R.3,Hunzicker-Dunn Mary12

Affiliation:

1. Department of Cell and Molecular Biology (A.B.K., E.T.M., M.P.F, J.C.J., M.H.-D.), Northwestern University Feinberg School of Medicine, Chicago, Illinois 60611

2. School of Molecular Biosciences (A.B.K., M.H.-D., E.A.S.), Washington State University, Pullman, Washington 99164

3. Department of Biochemistry and Molecular Biology (J.R.B.), Colorado State University, Fort Collins, Colorado 80523

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Reference103 articles.

1. Luteinizing hormone/choriogonadotropin-dependent, cholera toxin-catalyzed adenosine 5′-diphosphate (ADP)-ribosylation of the long and short forms of Gsα and pertussis toxin-catalyzed ADP-ribosylation of Giα.;Rajagopalan-Gupta;Mol Endocrinol,1997

2. Acute signaling by the LH receptor is independent of protein kinase C activation.;Salvador;Endocrinology,2002

3. Novel signaling pathways that control ovarian follicular development, ovulation, and luteinization.;Richards;Recent Prog Horm Res,2002

4. Luteinizing hormone-dependent activation of the epidermal growth factor network is essential for ovulation.;Hsieh;Mol Cell Biol,2007

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