Inflammation as a Sensor of Metabolic Stress in Obesity and Type 2 Diabetes
Author:
Affiliation:
1. Division of Endocrinology, Diabetes, and Metabolism and Department of Biomedicine, University Hospital Basel, CH-4031 Basel, Switzerland
Publisher
The Endocrine Society
Subject
Endocrinology
Link
http://academic.oup.com/endo/article-pdf/152/11/4005/9010845/endo4005.pdf
Reference17 articles.
1. Type 2 diabetes as an inflammatory disease.;Donath;Nat Rev Immunol,2011
2. Elimination of the NLRP3-ASC inflammasome protects against chronic obesity-induced pancreatic damage.;Youm;Endocrinology,2011
3. Free fatty acids induce a proinflammatory response in islets via the abundantly expressed interleukin-1 receptor I.;Böni-Schnetzler;Endocrinology,2009
4. Glucose-induced β-cell production of interleukin-1β contributes to glucotoxicity in human pancreatic islets.;Maedler;J Clin Invest,2002
5. Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes.;Masters;Nat Immunol,2010
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