Thyroid Hormone Receptor α and Regulation of Type 3 Deiodinase

Author:

Barca-Mayo Olga1,Liao Xiao-Hui1,Alonso Manuela1,Di Cosmo Caterina1,Hernandez Arturo2,Refetoff Samuel13,Weiss Roy E.13

Affiliation:

1. Departments of Medicine (O.B.-M., X.-H.L., M.A., C.D., S.R., R.E.W.)The University of Chicago, Chicago, Illinois 60637;

2. Dartmouth Medical School (A.H.), Lebanon, New Hampshire 03756

3. Pediatrics (S.R., R.E.W.), The University of Chicago, Chicago, Illinois 60637;

Publisher

The Endocrine Society

Subject

Endocrinology,Molecular Biology,General Medicine

Reference40 articles.

1. Differential and tissue-specific regulation of the multiple rat c-erbA messenger RNA species by thyroid hormone.;Hodin;J Clin Invest,1990

2. Identification of transcripts initiated from an internal promoter in the c-erbA α locus that encode inhibitors of retinoic acid receptor-α and triiodothyronine receptor activities.;Chassande;Mol Endocrinol,1997

3. Human carboxyl-terminal variant of α-type c-erbA inhibits trans-activation by thyroid hormone receptors without binding thyroid hormone.;Lazar;Proc Natl Acad Sci USA,1989

4. Dominant negative activity of thyroid hormone receptor variant α2 and interaction with nuclear corepressors.;Burgos-Trinidad;Mol Cell Endocrinol,1999

5. The thyroid hormone receptor variant α2 is a weak antagonist because it is deficient in interactions with nuclear receptor corepressors.;Tagami;Endocrinology,1998

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