Defective Transport of the Obesity Mutant PC1/3 N222D Contributes to Loss of Function-Reference-Cited by-同舟云学术

Defective Transport of the Obesity Mutant PC1/3 N222D Contributes to Loss of Function

Published:2014-07-01 Issue:7 Volume:155 Page:2391-2401
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Prabhu Yogikala¹,Blanco Elias H.¹,Liu Ming²,Peinado Juan R.¹,Wheeler Matthew C.³,Gekakis Nicholas³,Arvan Peter²,Lindberg Iris¹

Affiliation:

1. Department of Anatomy and Neurobiology (Y.P., E.H.B., J.R.P., I.L.), University of Maryland-Baltimore, Baltimore, Maryland 21201

2. Division of Endocrinology, Metabolism, and Diabetes (M.L., P.A.), University of Michigan, Michigan 48105

3. Department of Cell and Molecular Biology (M.C.W., N.G.), The Scripps Research Institute, San Diego, California 92037

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/155/7/2391/10826268/endo2391.pdf

Reference42 articles.

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3. Differential effects of temperature blockade on the proteolytic processing of three secretory granule-associated proteins;Milgram;J Cell Sci,1994

4. Comparative biosynthesis, covalent post-translational modifications and efficiency of prosegment cleavage of the prohormone convertases PC1 and PC2: glycosylation, sulphation and identification of the intracellular site of prosegment cleavage of PC1 and PC2;Benjannet;Biochem J,1993

5. Posttranslational maturation of the prohormone convertase sPC3 in vitro;Coates;J Neurochem,1997

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