Impaired Glucose-Stimulated Insulin Secretion, Enhanced Intraperitoneal Insulin Tolerance, and Increased β-Cell Mass in Mice Lacking the p110γ Isoform of Phosphoinositide 3-Kinase-Reference-Cited by-同舟云学术

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Impaired Glucose-Stimulated Insulin Secretion, Enhanced Intraperitoneal Insulin Tolerance, and Increased β-Cell Mass in Mice Lacking the p110γ Isoform of Phosphoinositide 3-Kinase

Published:2004-09 Issue:9 Volume:145 Page:4078-4083
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:Endocrinology

Author:

MacDonald P. E.,Joseph J. W.,Yau D.,Diao J.,Asghar Z.,Dai F.,Oudit G. Y.,Patel M. M.,Backx P. H.,Wheeler M. B.

Publisher

The Endocrine Society

Subject

Endocrinology

Cited by 44 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Hypertonicity during a rapid rise in D-glucose mediates first-phase insulin secretion;Frontiers in Endocrinology;2024-06-26

2. Vascular endothelial growth factor B inhibits insulin secretion in MIN6 cells and reduces Ca2+ and cyclic adenosine monophosphate levels through PI3K/AKT pathway;World Journal of Diabetes;2021-04-15

3. Bivariate genome-wide association study (GWAS) of body mass index and blood pressure phenotypes in northern Chinese twins;PLOS ONE;2021-02-04

4. Function, Regulation and Biological Roles of PI3Kγ Variants;Biomolecules;2019-08-30

5. p110γ deficiency protects against pancreatic carcinogenesis yet predisposes to diet-induced hepatotoxicity;Proceedings of the National Academy of Sciences;2019-07-02

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