GABAAand GABABreceptor dysregulation in superior frontal cortex of subjects with schizophrenia and bipolar disorder-Reference-Cited by-同舟云学术

GABAAand GABABreceptor dysregulation in superior frontal cortex of subjects with schizophrenia and bipolar disorder

Published:2017-04-05 Issue:7 Volume:71 Page:e21973
ISSN:0887-4476
Container-title:Synapse
language:en
Short-container-title:Synapse

Author:

Fatemi S. Hossein¹²^ORCID,Folsom Timothy D.¹,Thuras Paul D.³

Affiliation:

1. Department of Psychiatry; Division of Neuroscience Research, University of Minnesota Medical School; 420 Delaware St. SE, MMC 392 Minneapolis Minnesota 55455

2. Department of Neuroscience; University of Minnesota Medical School; 321 Church St. SE Minneapolis Minnesota 55455

3. Department of Psychiatry; VA Medical Center; 1 Veterans Drive Minneapolis Minnesota 55417-2399

Funder

National Institute of Mental Health

Ewald Bipolar Disease Research Fund

Bernstein Endowed Chair in Adult Psychiatry

PHS

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference68 articles.

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2. Gene expression for glutamic acid decarboxylase is reduced without loss of neurons in prefrontal cortex of schizophrenics;Akbarian;Archives of General Psychiatry,1995a

3. Rare variants in neuronal excitability genes influence risk for bipolar disorder;Ament;Proceedings of the National Academy of Sciences,2015

4. Inhibitory transmission in locus coeruleus neurons expressing GABAA receptor epsilon subunit has a number of unique properties;Belujon;Journal of Neurophysiology,2009

5. Differences in the subregional and cellular distribution of GABAA receptor binding in the hippocampal formation of schizophrenic brain;Benes;Synapse,1996b

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