Fyn kinase-regulated NMDA receptor- and AMPA receptor-dependent pain sensitization in spinal dorsal horn of mice
Author:
Affiliation:
1. Department of Molecular Pharmacology; School of Pharmacy; Lanzhou University; Gansu China
2. College of Chemistry and Chemical Engineering; Lanzhou University; Gansu China
Publisher
Wiley
Subject
Anesthesiology and Pain Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/j.1532-2149.2014.00455.x/fullpdf
Reference29 articles.
1. Fyn kinase-mediated phosphorylation of NMDA receptor NR2B subunit at Tyr1472 is essential for maintenance of neuropathic pain;Abe;Eur J Neurosci,2005
2. Perturbing PSD-95 interactions with NR2B-subtype receptors attenuates spinal nociceptive plasticity and neuropathic pain;D'Mello;Mol Ther,2011
3. Long term synaptic depression that is associated with GluR1 dephosphorylation but not alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor internalization;Davies;J Biol Chem,2008
4. Dopamine D1-dependent trafficking of striatal N-methyl-D-aspartate glutamate receptors requires Fyn protein tyrosine kinase but not DARPP-32;Dunah;Mol Pharmacol,2004
5. Impaired long-term potentiation, spatial learning, and hippocampal development in Fyn mutant mice;Grant;Science,1992
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