Modulation of inflammation by kininogen deficiency in a rat model of inflammatory arthritis-Reference-Cited by-同舟云学术

Modulation of inflammation by kininogen deficiency in a rat model of inflammatory arthritis

Published:2005 Issue:8 Volume:52 Page:2549-2552
ISSN:0004-3591
Container-title:Arthritis & Rheumatism
language:en
Short-container-title:Arthritis Rheum

Author:

Sainz Irma M.,Isordia-Salas Irma,Castaneda Julian L.,Agelan Alexis,Liu Bo,DeLa Cadena Raul A.,Pixley Robin A.,Adam Albert,Sartor R. Balfour,Colman Robert W.

Publisher

Wiley

Subject

Pharmacology (medical),Immunology,Rheumatology,Immunology and Allergy

Reference16 articles.

1. Modulation of the susceptibility of inbred and outbred rats to arthritis induced by cell walls of group A streptococci

2. , , , . Contact activation pathway: inflammatory, fibrinolytic, anticoagulant, antiadhesive, and antiangiogenic activities. In: editor. Hemostasis and thrombosis, basic principles and clinical practice. 4th ed. Philadelphia: Lippincott Williams & Wilkins; 2001. p. 103-21.

3. Induction of expression of monocyte interleukin 1 by Hageman factor (factor XII).

4. Heat shock protein 90 catalyzes activation of the prekallikrein-kininogen complex in the absence of factor XII

5. Recombinant prolylcarboxypeptidase activates plasma prekallikrein

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