Sensorimotor dysfunction in a mild mouse model of cortical contusion injury without significant neuronal loss is associated with increases in inflammatory proteins with innate but not adaptive immune functions-Reference-Cited by-同舟云学术

Sensorimotor dysfunction in a mild mouse model of cortical contusion injury without significant neuronal loss is associated with increases in inflammatory proteins with innate but not adaptive immune functions

Published:2020-12-02 Issue:6 Volume:99 Page:1533-1549
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Nieves Michael D.¹²³^ORCID,Furmanski Orion¹²,Doughty Martin L.¹²³^ORCID

Affiliation:

1. Center for Neuroscience and Regenerative Medicine Uniformed Services University of the Health Sciences Bethesda MD USA

2. Department of Anatomy, Physiology & Genetics, F.E. Hébert School of Medicine Uniformed Services University of the Health Sciences Bethesda MD USA

3. Graduate Program in Neuroscience, F.E. Hébert School of Medicine Uniformed Services University of the Health Sciences Bethesda MD USA

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24766

Reference67 articles.

1. Post-Injury Neuroprotective Effects of the Thalidomide Analog 3,6′-Dithiothalidomide on Traumatic Brain Injury

2. The adhesive removal test: a sensitive method to assess sensorimotor deficits in mice

3. Astrocyte roles in traumatic brain injury

4. Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration

5. Interleukin 1β and interleukin 6 relationship with paediatric head trauma severity and outcome

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