Caveolin‐1 Mediates Inflammatory Breast Cancer Cell Invasion via the Akt1 Pathway and RhoC GTPase
Author:
Affiliation:
1. Department of Biological Sciences, The Center for Translational Cancer ResearchThe University of DelawareNewarkDelaware
2. The Helen F. Graham Cancer CenterNewarkDelaware
Funder
Congressionally Directed Medical Research Programs
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.25025
Reference50 articles.
1. Breast cancer nodal metastasis correlates with tumour and lymph node methylation profiles of Caveolin-1 and CXCR4
2. The interaction between caveolin-1 and Rho-GTPases promotes metastasis by controlling the expression of alpha5-integrin and the activation of Src, Ras and Erk
3. Statin use in primary inflammatory breast cancer: a cohort study
4. Hsp90 and Caveolin Are Key Targets for the Proangiogenic Nitric Oxide–Mediated Effects of Statins
5. Farnesyl transferase inhibitor treatment of breast cancer cells leads to altered RhoA and RhoC GTPase activity and induces a dormant phenotype
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