Non-trisomic homeobox gene expression during craniofacial development in the Ts65Dn mouse model of Down syndrome

Author:

Billingsley Cherie N.1,Allen Jared R.1,Baumann Douglas D.2,Deitz Samantha L.1,Blazek Joshua D.1,Newbauer Abby1,Darrah Andrew1,Long Brad C.3,Young Brandon3,Clement Mark4,Doerge R.W.2,Roper Randall J.1

Affiliation:

1. Department of Biology and Indiana University Center for Regenerative Biology and Medicine; Indiana University-Purdue University Indianapolis; Indianapolis; Indiana

2. Department of Statistics; Purdue University; West Lafayette; Indiana

3. Genomics Core; Scripps Florida; Jupiter; Florida

4. Department of Computer Science; Brigham Young University; Provo; Utah

Publisher

Wiley

Subject

Genetics (clinical),Genetics

Reference54 articles.

1. Classification of human chromosome 21 gene-expression variations in Down syndrome: Impact on disease phenotypes;Ait Yahya-Graison;Am J Hum Genet,2007

2. Interactions between Sox9 and beta-catenin control chondrocyte differentiation;Akiyama;Genes Dev,2004

3. Anthropometric craniofacial pattern profiles in Down syndrome;Allanson;Am J Med Genet,1993

4. NFAT dysregulation by increased dosage of DSCR1 and DYRK1A on chromosome 21;Arron;Nature,2006

5. Targeted disruption of the Hoxb-2 locus in mice interferes with expression of Hoxb-1 and Hoxb-4;Barrow;Development,1996

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