Progranulin deficiency promotes persistent neuroinflammation and causes regional pathology in the hippocampus following traumatic brain injury
Author:
Affiliation:
1. Laboratory of Gene Therapy, Department of Biochemistry College of Life Sciences, Shaanxi Normal University Xi'an Shaanxi China
2. Department of Pathology University of Utah Salt Lake City Utah USA
Funder
Fundamental Research Funds for the Central Universities
National Natural Science Foundation of China
Illinois Department of Public Health
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Neurology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.24175
Reference84 articles.
1. Activated microglia impairs neuroglial interaction by opening Cx43 hemichannels in hippocampal astrocytes
2. Absence of TLR4 Reduces Neurovascular Unit and Secondary Inflammatory Process after Traumatic Brain Injury in Mice
3. Progranulin, a Glycoprotein Deficient in Frontotemporal Dementia, Is a Novel Substrate of Several Protein Disulfide Isomerase Family Proteins
4. Glial influence on the blood brain barrier
5. Mutations in progranulin cause tau-negative frontotemporal dementia linked to chromosome 17
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