Bcr‐Abl activates AURKA and AURKB in chronic myeloid leukemia cells via AKT signaling
Author:
Affiliation:
1. Department of Hematology and Respiratory MedicineKochi UniversityNankoku Kochi Japan
2. Department of ImmunologyKochi Medical School, Kochi University, Kochi UniversityNankoku Kochi Japan
Publisher
Wiley
Subject
Cancer Research,Oncology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/ijc.28434
Reference37 articles.
1. Molecular mechanisms of transformation by the BCR-ABL oncogene
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3. Cell lines and clinical isolates derived from Ph1-positive chronic myelogenous leukemia patients express c-abl proteins with a common structural alteration.
4. Genetic requirement for Ras in the transformation of fibroblasts and hematopoietic cells by the Bcr-Abl oncogene.
5. Transformation of hematopoietic cells by BCR/ABL requires activation of a PI-3k/Akt-dependent pathway
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