The MTNR1A mRNA is stabilized by the cytoplasmic hnRNPL in renal tubular cells

Author:

Huang Yen‐Sung1ORCID,Lu Kuo‐Cheng2,Chao Hsu‐Wen3ORCID,Chen Ann4,Chao Tai‐Kuang4,Guo Cheng‐Yi5,Hsieh Hsin‐Yi5,Shih Hsiu‐Ming16,Sytwu Huey‐Kang78,Wu Chia‐Chao58ORCID

Affiliation:

1. Institute of Biomedical Sciences Academia Sinica Taipei Taiwan

2. Division of Nephrology, Department of Medicine, Taipei Tzu Chi Hospital Buddhist Tzu Chi Medical Foundation New Taipei City Taiwan

3. Department of Physiology, School of Medicine, College of Medicine Taipei Medical University Taipei Taiwan

4. Department of Pathology, Tri‐Service General Hospital National Defense Medical Center Taipei Taiwan

5. Division of Nephrology, Department of Internal Medicine, Tri‐Service General Hospital National Defense Medical Center Taipei Taiwan

6. Institute of Molecular and Genomic Medicine National Health Research Institutes Miaoli County Taiwan

7. National Institute of Infectious Diseases and Vaccinology National Health Research Institutes Miaoli County Taiwan

8. Department and Graduate Institute of Microbiology and Immunology National Defense Medical Center Taipei Taiwan

Funder

Ministry of Science and Technology

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Reference40 articles.

1. Melatonin MT1 and MT2 receptor expression in Parkinson's disease;Adi N.;Medical Science Monitor,2010

2. Regulation of Pituitary MT1 Melatonin Receptor Expression by Gonadotrophin-Releasing Hormone (GnRH) and Early Growth Response Factor-1 (Egr-1): In Vivo and In Vitro Studies

3. Regulation of the Mel 1a melatonin receptor mRNA and protein levels in the ovine pars tuberalis: Evidence for a cyclic adenosine 3',5'‐monophosphate‐independent Mel 1a receptor coupling and an autoregulatory mechanism of expression;Barrett P.;Molecular Endocrinology,1996

4. Melatonin receptors: molecular pharmacology and signalling in the context of system bias

5. Global analysis of physical and functional RNA targets of hnRNP L reveals distinct sequence and epigenetic features of repressed and enhanced exons

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