BOK displays cell death‐independent tumor suppressor activity in non‐small‐cell lung carcinoma-Reference-Cited by-同舟云学术

BOK displays cell death‐independent tumor suppressor activity in non‐small‐cell lung carcinoma

Published:2017-08-07 Issue:10 Volume:141 Page:2050-2061
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Moravcikova Erika¹²,Krepela Evzen³,Donnenberg Vera S.²,Donnenberg Albert D.⁴,Benkova Kamila⁵,Rabachini Tatiana¹,Fernandez‐Marrero Yuniel¹,Bachmann Daniel¹,Kaufmann Thomas¹^ORCID

Affiliation:

1. Institute of Pharmacology, Faculty of Medicine, University of BernBern Switzerland

2. Department of Cardiothoracic SurgerySchool of Medicine, University of Pittsburgh PA

3. Institute of Biochemistry and Experimental Oncology, First Faculty of Medicine, Charles UniversityPrague Czech Republic

4. Department of MedicineSchool of Medicine, University of Pittsburgh PA

5. Department of PathologyHospital BulovkaPrague Czech Republic

Funder

U.S. Department of Defense

National Cancer Institute

Krebsliga Schweiz

Swiss National Science Foundation

Publisher

Wiley

Subject

Cancer Research,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ijc.30906

Reference42 articles.

1. BOK Is a Non-canonical BCL-2 Family Effector of Apoptosis Regulated by ER-Associated Degradation

2. Intracellular localization of the BCL-2 family member BOK and functional implications

3. Bok is a pro-apoptotic Bcl-2 protein with restricted expression in reproductive tissues and heterodimerizes with selective anti-apoptotic Bcl-2 family members

4. Mtd, a Novel Bcl-2 Family Member Activates Apoptosis in the Absence of Heterodimerization with Bcl-2 and Bcl-XL

5. Bok is a genuine multi-BH-domain protein that triggers apoptosis in the absence of Bax and Bak and augments drug response

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