Desmin-related neuromuscular disorders
Author:
Publisher
Wiley
Subject
Physiology (medical),Cellular and Molecular Neuroscience,Neurology (clinical),Physiology
Reference104 articles.
1. : Cytoplasmic body myopathy: a dominantly inherited Japanese kindred [abstract]. Muscle Nerve 1994; (suppl 1): 4-17, S89.
2. Immunocytochemical localization of desmin at human neuromuscular junctions
3. Immunocytochemical localization of ubiquitin in inclusion body myositis allows its light‐microscopic distinction from polymyositis
4. Strong immunoreactivity of β-amyloid precursor protein, including the β-amyloid protein sequence, at human neuromuscular junctions
5. Prion protein is strongly immunolocalized at the postsynaptic domain of human normal neuromuscular junctions
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1. The FLNC Ala1186Val Variant Linked to Cytoplasmic Body Myopathy and Cardiomyopathy Causes Protein Instability;Biomedicines;2024-01-30
2. The Myocardial Accumulation of Aggregated Desmin Protein in a Case of Desminopathy with a <i>de novo DES</i> p.R406W Mutation;Internal Medicine;2023-10-01
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4. Desmin mutations impact the autophagy flux in C2C12 cell in mutation-specific manner;Cell and Tissue Research;2023-06-06
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