Drug Discovery for Pemphigoid Diseases
Author:
Affiliation:
1. Lübeck Institute of Experimental Dermatology, University of Lübeck; Lübeck Germany
Funder
Deutsche Forschungsgemeinschaft
Publisher
Wiley
Subject
General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/cpph.55/fullpdf
Reference47 articles.
1. In vitro and in vivo models to investigate the pathomechanisms and novel treatments for pemphigoid diseases;Bieber;Experimental Dermatology,2017
2. T cells mediate autoantibody-induced cutaneous inflammation and blistering in epidermolysis bullosa acquisita;Bieber;Scientific Reports,2016
3. The cartilage matrix protein subdomain of type VII collagen is pathogenic for epidermolysis bullosa acquisita;Chen;American Journal of Pathology,2007
4. Blister-inducing antibodies target multiple epitopes on collagen VII in mice;Csorba;Journal of Cellular and Molecular Medicine,2014
5. Skin microbiota-associated inflammation precedes autoantibody induced tissue damage in experimental epidermolysis bullosa acquisita;Ellebrecht;Journal of Autoimmunity,2016
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2. C-type lectin receptor expression is a hallmark of neutrophils infiltrating the skin in epidermolysis bullosa acquisita;Frontiers in Immunology;2023-09-20
3. Pemphigoid diseases in older adults;Die Dermatologie;2023-08-18
4. The complement receptor C5aR2 regulates neutrophil activation and function contributing to neutrophil-driven epidermolysis bullosa acquisita;Frontiers in Immunology;2023-05-19
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