Disruption of dystonin in Schwann cells results in late‐onset neuropathy and sensory ataxia

Author:

Horie Masao123,Yoshioka Nozomu1,Kusumi Satoshi2,Sano Hiromi45,Kurose Masayuki67,Watanabe‐Iida Izumi89,Hossain Ibrahim110,Chiken Satomi45,Abe Manabu811,Yamamura Kensuke6,Sakimura Kenji811,Nambu Atsushi45,Shibata Masahiro2,Takebayashi Hirohide112ORCID

Affiliation:

1. Division of Neurobiology and AnatomyGraduate School of Medical and Dental Sciences, Niigata University Niigata Japan

2. Department of Morphological SciencesKagoshima University Kagoshima Japan

3. Department of NursingNiigata College of Nursing Niigata Japan

4. Division of System NeurophysiologyNational Institute for Physiological Sciences Okazaki Japan

5. Department of Physiological SciencesSOKENDAI Okazaki Japan

6. Division of Oral PhysiologyGraduate School of Medical and Dental Sciences, Niigata University Niigata Japan

7. Department of Physiology, School of Dentistry, Iwate Medical University Morioka Japan

8. Department of Cellular NeurobiologyBrain Research Institute, Niigata University Niigata Japan

9. Division of Oral Biochemistry, Graduate School of Medical and Dental Sciences, Niigata University Niigata Japan

10. Department of Biochemistry and Molecular BiologyJahangirnagar University Savar Dhaka Bangladesh

11. Department of Animal Model DevelopmentBrain Research Institute, Niigata University Niigata Japan

12. Center for Coordination of Research FacilitiesNiigata University Niigata Japan

Funder

Ministry of Education, Culture, Sports, Science and Technology

National Institute for Physiological Sciences

Takeda Medical Research Foundation

Uehara Memorial Foundation

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

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