Ablation of Hepatocyte Smad1 , Smad5 , and Smad8 Causes Severe Tissue Iron Loading and Liver Fibrosis in Mice-Reference-Cited by-同舟云学术

Ablation of Hepatocyte Smad1 , Smad5 , and Smad8 Causes Severe Tissue Iron Loading and Liver Fibrosis in Mice

Published:2019-08-13 Issue:6 Volume:70 Page:1986-2002
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Wang Chia‐Yu¹^ORCID,Xiao Xia¹,Bayer Abraham¹,Xu Yang¹,Dev Som¹,Canali Susanna¹,Nair Anil V.¹,Masia Ricard²,Babitt Jodie L.¹

Affiliation:

1. Program in Anemia Signaling Research, Division of Nephrology, Program in Membrane Biology, Center for Systems Biology Massachusetts General Hospital, Harvard Medical School Boston MA

2. Department of Pathology Massachusetts General Hospital, Harvard Medical School Boston MA

Funder

National Institutes of Health

Cooley's Anemia Foundation

Publisher

Wiley

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.30780

Reference40 articles.

1. Hepcidin Regulates Cellular Iron Efflux by Binding to Ferroportin and Inducing Its Internalization

2. Mutant antimicrobial peptide hepcidin is associated with severe juvenile hemochromatosis

3. Haemochromatosis

4. Liver iron sensing and body iron homeostasis

5. Endothelial cells produce bone morphogenetic protein 6 required for iron homeostasis in mice

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