Recovery from hypoxia‐induced internalization of cardiac Na + /H + exchanger 1 requires an adequate intracellular store of antioxidants
Author:
Affiliation:
1. Department of Biophysics Faculty of Medicine, Ankara University Ankara Turkey
2. Department of Physiology, Anatomy, and Genetics Oxford University Oxford UK
Funder
British Heart Foundation
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.27268
Reference50 articles.
1. The effects of changes of pH on intracellular calcium transients in mammalian cardiac muscle.
2. Investigating Mitochondrial Metabolism in Contracting HL-1 Cardiomyocytes Following Hypoxia and Pharmacological HIF Activation Identifies HIF-Dependent and Independent Mechanisms of Regulation
3. HL-1 mouse cardiomyocyte injury and death after simulated ischemia and reperfusion: roles of pH, Ca2+-independent phospholipase A2, and Na+/H+exchange
4. A Novel Hypoxia-inducible Factor-independent Hypoxic Response Regulating Mammalian Target of Rapamycin and Its Targets
5. Na+/h+exchange inhibitors for cardioprotective therapy: progress, problems and prospects
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1. Differential role of prolyl hydroxylases on mitochondrial function of HL-1 cells in a model of iron deficiency;Mitochondrion;2022-03
2. Dihydroartemisinin prompts amplification of photodynamic therapy-induced reactive oxygen species to exhaust Na/H exchanger 1-mediated glioma cells invasion and migration;Journal of Photochemistry and Photobiology B: Biology;2021-06
3. Cardiomyocyte Na+/H+ Exchanger-1 Activity Is Reduced in Hypoxia;Frontiers in Cardiovascular Medicine;2021-01-27
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