Resveratrol Reduces COMPopathy in Mice Through Activation of Autophagy
Author:
Affiliation:
1. Department of Pediatrics, McGovern Medical School The University of Texas Health Science Center at Houston (UTHealth) Houston TX USA
2. UTHealth School of Dentistry Houston TX USA
Funder
Center for Scientific Review
Publisher
Wiley
Subject
Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbm4.10456
Reference67 articles.
1. Nosology and classification of genetic skeletal disorders: 2019 revision
2. Mutations in exon 17B of cartilage oligomeric matrix protein (COMP) cause pseudoachondroplasia
3. Antioxidant and anti-inflammatory agents mitigate pathology in a mouse model of pseudoachondroplasia
4. Novel mTORC1 Mechanism Suggests Therapeutic Targets for COMPopathies
5. Chop (Ddit3) Is Essential for D469del-COMP Retention and Cell Death in Chondrocytes in an Inducible Transgenic Mouse Model of Pseudoachondroplasia
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