Deletion of Alzheimer's disease‐associated CD33 results in an inflammatory human microglia phenotype-Reference-Cited by-同舟云学术

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Deletion of Alzheimer's disease‐associated CD33 results in an inflammatory human microglia phenotype

Published:2021-02-04 Issue:6 Volume:69 Page:1393-1412
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Wißfeld Jannis¹,Nozaki Ichiro¹²,Mathews Mona¹³,Raschka Tamara⁴,Ebeling Christian⁴,Hornung Veit⁵,Brüstle Oliver¹³,Neumann Harald¹^ORCID

Affiliation:

1. Institute of Reconstructive Neurobiology University of Bonn Medical Faculty and University Hospital Bonn Bonn Germany

2. Department of Neurology and Neurobiology of Aging Kanazawa University Graduate School of Medical Sciences Kanazawa Japan

3. LIFE & BRAIN GmbH, Cellomics Unit Bonn Germany

4. Department of Bioinformatics Fraunhofer Institute for Algorithms and Scientific Computing Sankt Augustin Germany

5. Institute of Molecular Medicine University of Bonn Medical Faculty and University Hospital Bonn Bonn Germany

Funder

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23968

Reference67 articles.

1. High Resolution Crystal Structures of Siglec-7

2. The Membrane-Proximal Immunoreceptor Tyrosine-Based Inhibitory Motif Is Critical for the Inhibitory Signaling Mediated by Siglecs-7 and -9, CD33-Related Siglecs Expressed on Human Monocytes and NK Cells

3. Associations Between Genetic Variants in 19p13 and 19q13 Regions and Susceptibility to Alzheimer Disease: A Meta-Analysis

4. Repression of phagocytosis by human CD33 is not conserved with mouse CD33

5. Src-family kinases play an essential role in differentiation signaling downstream of macrophage colony-stimulating factor receptors mediating persistent phosphorylation of phospholipase C-γ2 and MAP kinases ERK1 and ERK2

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