Breaksinvisibleto the DNA damage response machinery accumulate in ATM-deficient cells
Author:
Publisher
Wiley
Subject
Cancer Research,Genetics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/gcc.20679/fullpdf
Reference40 articles.
1. Guiding ATM to Broken DNA;Abraham;Science,2005
2. Fibroblasts from ataxia telangiectasia (AT) and AT heterozygotes show an enhanced level of residual DNA double-strand breaks after low dose-rate gamma-irradiation as assayed by pulsed field gel electrophoresis;Blöcher;Int J Radiat Biol,1991
3. ATM stabilizes DNA double-strand-break complexes during V(D)J recombination;Bredemeyer;Nature,2006
4. Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining;Bredemeyer;J Immunol,2008
5. ATM phosphorylates histone H2AX in response to DNA double-strand breaks;Burma;J Biol Chem,2001
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5. p38 MAPK inhibits nonsense-mediated RNA decay in response to persistent DNA damage in noncycling cells;Journal of Biological Chemistry;2017-09
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