Inhibition of chemotactic factor-induced neutrophil responsiveness by arachidonic acid
Author:
Publisher
Wiley
Subject
Cell Biology,Clinical Biochemistry,Physiology
Reference35 articles.
1. Calmodulin antagonists modulate rabbit neutrophil degranulation, aggregation and stimulated oxygen consumption
2. Transformation of arachidonic acid by rabbit polymorphonuclear leukocytes. Formation of a novel dihydroxyeicosatetraenoic acid.
3. Chemoattractants stimulate degradation of methylated phospholipids and release of arachidonic acid in rabbit leukocytes.
4. Effects of free fatty acids on the organization of cytoskeletal elements in lymphocytes.
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1. Cyclosporin H is a potent and selective competitive antagonist of human basophil activation by N-formyl-methionyl-leucyl-phenylalanine☆, ☆☆, ★, ★★;Journal of Allergy and Clinical Immunology;1996-07
2. The chymotrypsin inhibitor carbobenzyloxy-leucine-tyrosine-chloromethylketone interferes with phospholipase D activation induced by formyl-methionyl-leucyl-phenylalanine in human neutrophils;Journal of Biological Chemistry;1991-08
3. Arachidonic acid-induced mobilization of calcium in human neutrophils: evidence for a multicomponent mechanism of action;British Journal of Pharmacology;1989-06
4. Arachidonic acid and 15(S)-hydroxy-5,8,11-cis-13-trans-eicosatetraenoic acid modulate human polymorphonuclear neutrophil activation by monocyte derived neutrophil activating factor;Biochemical and Biophysical Research Communications;1987-10
5. Arachidonic acid-induced release of calcium in permeabilized human neutrophils;FEBS Letters;1987-09-14
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