Fanconi anemia (FA) and crosslinker sensitivity: Re-appraising the origins of FA definition-Reference-Cited by-同舟云学术

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Fanconi anemia (FA) and crosslinker sensitivity: Re-appraising the origins of FA definition

Published:2015-03-02 Issue:7 Volume:62 Page:1137-1143
ISSN:1545-5009
Container-title:Pediatric Blood & Cancer
language:en
Short-container-title:Pediatr Blood Cancer

Author:

Pagano Giovanni¹,d'Ischia Marco²,Pallardó Federico V.³

Affiliation:

1. Istituto Nazionale Tumori Fondazione G. Pascale-Cancer Research Center at Mercogliano (CROM); Mercogliano (AV) Italy

2. Department of Chemical Sciences; University of Naples “Federico II,”; Naples Italy

3. University of Valencia-INCLIVA; CIBERER (Centro de Investigación Biomédica en Red de Enfermedades Raras); Valencia Spain

Publisher

Wiley

Subject

Oncology,Hematology,Pediatrics, Perinatology, and Child Health

Reference98 articles.

1. A high susceptibility of Fanconi's anemia to chromosome breakage by DNA cross-linking agents;Sasaki;Cancer Res,1973

2. Is Fanconi's anaemia defective in a process essential to the repair of DNA cross links;Sasaki;Nature,1975

3. Repair of mitomycin C damage to DNA in mammalian cells and its impairment in Fanconi's anemia cells;Fujiwara;Biochem Biophys Res Commun,1975

4. Defective repair of mitomycin C crosslinks in Fanconi's anemia and loss in confluent normal human and xeroderma pigmentosum cells;Fujiwara;Biochim Biophys Acta,1982

5. Induction by alkylating agents of sister chromatid exchanges and chromatid breaks in Fanconi's anemia;Latt;Proc Natl Acad Sci USA,1975

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