Improved antisense oligonucleotide induced exon skipping in themdx mouse model of muscular dystrophy-Reference-Cited by-同舟云学术

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Improved antisense oligonucleotide induced exon skipping in themdx mouse model of muscular dystrophy

Published:2002 Issue:6 Volume:4 Page:644-654
ISSN:1099-498X
Container-title:The Journal of Gene Medicine
language:en
Short-container-title:J. Gene Med.

Author:

Mann Christopher J.,Honeyman Kaite,McClorey Graham,Fletcher Sue,Wilton Stephen D.

Publisher

Wiley

Subject

Genetics (clinical),Drug Discovery,Genetics,Molecular Biology,Molecular Medicine

Reference48 articles.

1. Dystrophin: The protein product of the duchenne muscular dystrophy locus

2. Characterization of Dystrophin in Muscle-Biopsy Specimens from Patients with Duchenne's or Becker's Muscular Dystrophy

3. An explanation for the phenotypic differences between patients bearing partial deletions of the DMD locus

4. Cellular and humoral immune responses to adenoviral vectors containing factor IX gene: tolerization of factor IX and vector antigens allows for long-term expression.

5. Hypothesis: naked plasmid DNA is taken up by cellsin vivo by a receptor-mediated process

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1. RNA-Based Strategies for Cancer Therapy: In Silico Design and Evaluation of ASOs for Targeted Exon Skipping;International Journal of Molecular Sciences;2023-10-03

2. Next Generation Exon 51 Skipping Antisense Oligonucleotides for Duchenne Muscular Dystrophy;Nucleic Acid Therapeutics;2023-06-01

3. Injectable hydrogel microspheres for sustained gene delivery of antisense oligonucleotides to restore the expression of dystrophin protein in duchenne muscular dystrophy;European Polymer Journal;2022-03

4. 7′,5′-alpha-bicyclo-DNA: new chemistry for oligonucleotide exon splicing modulation therapy;Nucleic Acids Research;2021-11-26

5. Intramuscular Evaluation of Chimeric Locked Nucleic Acid/2′OMethyl-Modified Antisense Oligonucleotides for Targeted Exon 23 Skipping in Mdx Mice;Pharmaceuticals;2021-10-30

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