Promyelocytic leukemia protein deficiency leads to spontaneous formation of liver tumors in hepatitis C virus transgenic mice-Reference-Cited by-同舟云学术

Promyelocytic leukemia protein deficiency leads to spontaneous formation of liver tumors in hepatitis C virus transgenic mice

Published:2019-05-29 Issue:8 Volume:8 Page:3793-3802
ISSN:2045-7634
Container-title:Cancer Medicine
language:en
Short-container-title:Cancer Med

Author:

Straub Katja¹,Husen Peri²,Baba Hideo A.³,Trippler Martin¹,Wedemeyer Heiner¹,Herzer Kerstin¹^ORCID

Affiliation:

1. Department of Gastroenterology and Hepatology, Faculty of Medicine, University Hospital Essen University of Duisburg‐Essen Essen Germany

2. Department of General‐, Visceral‐ and Transplantation Surgery, Faculty of Medicine, University Hospital Essen University of Duisburg‐Essen Essen Germany

3. Institute of Pathology, Faculty of Medicine, University Hospital Essen University of Duisburg‐Essen Essen Germany

Funder

Deutsche Krebshilfe

Deutsche Forschungsgemeinschaft

Publisher

Wiley

Subject

Cancer Research,Radiology, Nuclear Medicine and imaging,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cam4.2162

Reference21 articles.

1. Hepatocellular carcinoma

2. Hepatitis C virus-induced hepatocellular carcinoma

3. Nonalcoholic steatohepatitis is the most rapidly growing indication for liver transplantation in patients with hepatocellular carcinoma in the U.S.

4. Global prevalence and genotype distribution of hepatitis C virus infection in 2015: a modelling study

5. Hepatocellular carcinoma: a global view

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