Experimental study on protection of vitamin B6on TCDD-induced palatal cleft formation in the mice
Author:
Publisher
Wiley
Subject
Health, Toxicology and Mutagenesis,Developmental Biology,Toxicology,Embryology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/bdrb.20203/fullpdf
Reference28 articles.
1. Review of the interaction between TCDD and glucocorticoids in embryonic palate;Abbott;Toxicology,1995
2. Comparisons of the effects of TCDD and hydrocortisone on growth factor expression provide insight into their interaction in the embryonic mouse palate;Abbott;Teratology,1992
3. AHR, ARNT, and CYP1A1 Mrna quantitation in cultured human embryonic palates exposed to TCDD and comparison with mouse palate in vivo and in culture;Abbott;Toxicol Sci,1999
4. EGF and TGF-α expression influence the developmental toxicity of TCDD: Dose response and AHR phenotype in EGF, TGF-α, and EGF+TGF-α knockout mice;Abbott;Toxicol Sci,2003
5. Protective effects of vitamin A and vitamin E succinate against 2.3.7.8.-tetrachlorodibenzo-p-dioxin (TCDD)-induced body wasting, hepatomegaly, thymic atrophy, production of reactive oxygen species and DNA damage in C57BL/6J mice;Alsharif;Basic Clin Pharmacol Toxicol,2004
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1. Genetic background and window of exposure contribute to thyroid dysfunction promoted by low-dose exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin in mice;Scientific Reports;2018-11-05
2. Prevalence of Dental Anomalies in Patients With Cleft Lip and Palate;Journal of Craniofacial Surgery;2013-09
3. Palm and Finger Print Characteristics in Nonfamilial Cleft Lip and Palate Patients and their Parents;Journal of Craniofacial Surgery;2013-05
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