Meta-analysis of hypercoagulability genetic polymorphisms in perthes disease

Author:

Woratanarat Patarawan1,Thaveeratitharm Charnwit1,Woratanarat Thira2,Angsanuntsukh Chanika1,Attia John3,Thakkinstian Ammarin4

Affiliation:

1. Department of Orthopaedics; Faculty of Medicine Ramathibodi Hospital, Mahidol University; 270 Rama VI Road, Ratchathewee Bangkok 10400 Thailand

2. Department of Preventive and Social Medicine; Faculty of Medicine, Chulalongkorn University; Bangkok 10330 Thailand

3. Centre for Clinical Epidemiology and Biostatistics, The University of Newcastle; Newcastle NSW Australia

4. Section for Clinical Epidemiology and Biostatistics; Faculty of Medicine Ramathibodi Hospital, Mahidol University; Bangkok 10400 Thailand

Publisher

Wiley

Subject

Orthopedics and Sports Medicine

Reference29 articles.

1. Protein C and S deficiency, thrombophilia, and hypofibrinolysis: pathophysiologic causes of Legg-Perthes disease;Glueck;Pediatr Res,1994

2. Perthes' disease and the search for genetic associations: collagen mutations, Gaucher's disease and thrombophilia;Kenet;J Bone Joint Surg Br,2008

3. Secondhand smoke, hypofibrinolysis, and Legg-Perthes disease;Glueck;Clin Orthop Relat Res,1998

4. The plasminogen activator inhibitor-1 gene, hypofibrinolysis, and osteonecrosis;Glueck;Clin Orthop Relat Res,1999

5. Inherited resistance to activated protein C caused by presence of the FV:Q506 allele as a basis of venous thrombosis;Dahlback;Haemostasis,1996

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