Profound defects inβ-cell function in screen-detected type 2 diabetes are not improved with glucose-lowering treatment in the Early Diabetes Intervention Program (EDIP)
Author:
Affiliation:
1. Indiana University School of Medicine; Indianapolis IN USA
2. University of North Carolina; Chapel Hill NC USA
3. St Joseph Mercy Hospital; Ann Arbor MI USA
Funder
National Institutes of Health
Bayer Healthcare
Publisher
Wiley
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism,Internal Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/dmrr.2553/fullpdf
Reference26 articles.
1. The relative contributions of insulin resistance and beta-cell dysfunction to the pathophysiology of type 2 diabetes;Kahn;Diabetologia,2003
2. Islet beta cell failure in type 2 diabetes;Prentki;J Clin Invest,2006
3. Treating postprandial hyperglycemia does not appear to delay progression of early type 2 diabetes: the early diabetes intervention program;Kirkman;Diabetes Care,2006
4. A study of the effects of acarbose on glucose metabolism in patients predisposed to developing diabetes: the Dutch acarbose intervention study in persons with impaired glucose tolerance (DAISI);Nijpels;Diabetes Metab Res Rev,2008
5. Glucose clamp technique: a method for quantifying insulin secretion and resistance;DeFronzo;Am J Physiol,1979
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