Bone Matrix Mineralization and Response to Burosumab in Adult Patients With X‐Linked Hypophosphatemia: Results From the Phase 3, Single‐Arm International Trial
Author:
Affiliation:
1. Ludwig Boltzmann Institute of Osteology at Hanusch Hospital of OEGK and AUVA Trauma Centre Meidling 1st Med Department Hanusch Hospital Vienna Austria
2. Vienna Bone and Growth Center Vienna Austria
Funder
Ultragenyx Pharmaceutical
Allgemeine Unfallversicherungsanstalt
Publisher
Wiley
Subject
Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.4641
Reference80 articles.
1. A clinician's guide to X-linked hypophosphatemia
2. Proteolytic processing of osteopontin by PHEX and accumulation of osteopontin fragments in Hyp mouse bone, the murine model of X-linked hypophosphatemia
3. Abnormal osteopontin and matrix extracellular phosphoglycoprotein localization, and odontoblast differentiation, in X-linked hypophosphatemic teeth
4. Biological stenciling of mineralization in the skeleton: Local enzymatic removal of inhibitors in the extracellular matrix
5. FGF23 and its role in X-linked hypophosphatemia-related morbidity
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1. Real‐world data of Brazilian adults with X‐linked hypophosphatemia (XLH) treated with burosumab and comparison with other worldwide cohorts;Molecular Genetics & Genomic Medicine;2024-02
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3. Dental impact of anti-fibroblast growth factor 23 therapy in X-linked hypophosphatemia;International Journal of Oral Science;2023-12-06
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