TGF-β transactivates EGFR and facilitates breast cancer migration and invasion through canonical Smad3 and ERK/Sp1 signaling pathways-Reference-Cited by-同舟云学术

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TGF-β transactivates EGFR and facilitates breast cancer migration and invasion through canonical Smad3 and ERK/Sp1 signaling pathways

Published:2018-01-24 Issue:3 Volume:12 Page:305-321
ISSN:1574-7891
Container-title:Molecular Oncology
language:en
Short-container-title:Mol Oncol

Author:

Zhao Yuanyuan¹²³⁴,Ma Jing¹²³⁴,Fan Yanling¹²³⁴,Wang Zhiyong¹²³⁴,Tian Ran¹²³⁴,Ji Wei¹²³⁴,Zhang Fei¹²³⁴,Niu Ruifang¹²³⁴

Affiliation:

1. Public Laboratory; National Clinical Research Center for Cancer; Tianjin Medical University Cancer Institute and Hospital; China

2. Key Laboratory of Cancer Prevention and Therapy; Tianjin China

3. Tianjin's Clinical Research Center for Cancer; China

4. Key Laboratory of Breast Cancer Prevention and Therapy; Ministry of Education; Tianjin Medical University; China

Funder

National Natural Science Foundation of China

Tianjin Municipal Science and Technology Commission

Changjiang Scholars and Innovative Research Team

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Reference44 articles.

1. Expression and co-expression of the members of the epidermal growth factor receptor (EGFR) family in invasive breast carcinoma;Abd El-Rehim;Br J Cancer,2004

2. ERBB receptors: from oncogene discovery to basic science to mechanism-based cancer therapeutics;Arteaga;Cancer Cell,2014

3. Histone deacetylase inhibitors upregulate MMP11 gene expression through Sp1/Smad complexes in human colon adenocarcinoma cells;Barrasa;Biochem Biophys Acta,2012

4. Inhibition of TGF-beta enhances the in vivo antitumor efficacy of EGF receptor-targeted therapy;Bedi;Mol Cancer Ther,2012

5. Transforming growth factor beta as regulator of cancer stemness and metastasis;Bellomo;Br J Cancer,2016

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