Mutations ofFLT3,NRAS,KRAS, andPTPN11 are frequent and possibly mutually exclusive in high hyperdiploid childhood acute lymphoblastic leukemia-Reference-Cited by-同舟云学术

Mutations ofFLT3,NRAS,KRAS, andPTPN11 are frequent and possibly mutually exclusive in high hyperdiploid childhood acute lymphoblastic leukemia

Published:2007 Issue:1 Volume:47 Page:26-33
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosom. Cancer

Author:

Paulsson Kajsa,Horvat Andrea,Strömbeck Bodil,Nilsson Fredrik,Heldrup Jesper,Behrendtz Mikael,Forestier Erik,Andersson Anna,Fioretos Thoas,Johansson Bertil

Publisher

Wiley

Subject

Cancer Research,Genetics

Reference44 articles.

1. Clinical impact of internal tandem duplications and activating point mutations in FLT3 in acute myeloid leukemia in elderly patients

2. Molecular signatures in childhood acute leukemia and their correlations to expression patterns in normal hematopoietic subpopulations

3. Fluorescence in situ hybridization analyses of hematologic malignancies reveal frequent cytogenetically unrecognized 12p rearrangements

4. Cytogenetic and FISH studies of a single center consecutive series of 152 childhood acute lymphoblastic leukemias

5. FLT3 mutations in childhood acute lymphoblastic leukemia

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